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mouse monoclonal anti ctip  (Santa Cruz Biotechnology)


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    Structured Review

    Santa Cruz Biotechnology mouse monoclonal anti ctip
    Mouse Monoclonal Anti Ctip, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 91 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+monoclonal+anti+ctip/CtIP+Antibody/pm40720252-82-23-53
    Average 93 stars, based on 91 article reviews
    mouse monoclonal anti ctip - by Bioz Stars, 2026-11
    93/100 stars

    Images

    Related Articles

    Recombinant:

    Article Title: Human RIF1-Protein Phosphatase 1 Prevents Degradation and Breakage of Nascent DNA on Replication Stalling
    Article Snippet: Mouse monoclonal anti-CtIP [D-4] , Santa Cruz , Cat# sc-271339; RRID: AB_10608728.

    Article Title: Discovery of Bi-magnolignan as a novel BRD4 inhibitor inducing apoptosis and DNA damage for cancer therapy.
    Article Snippet: Bi-magnolignan (BM), a novel compound isolated from Magnolia Officinalis leaves, exhibits significant anti-tumor activity in vitro.. However, the underlying mechanism remains elusive.. This study examines the anti-tumor properties of BM and its mechanism of action, specifically through its interaction with BRD4, a key regulator in oncogene transcription and genome stability.

    Article Title: β-TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1.
    Article Snippet: Samples were analyzed by MS/MS using an LTQ mass spectrometer (Thermo Scientific, Waltham, MA, USA) as described previously [21].

    Imaging:

    Article Title: Human RIF1-Protein Phosphatase 1 Prevents Degradation and Breakage of Nascent DNA on Replication Stalling
    Article Snippet: Mouse monoclonal anti-CtIP [D-4] , Santa Cruz , Cat# sc-271339; RRID: AB_10608728.

    Article Title: Discovery of Bi-magnolignan as a novel BRD4 inhibitor inducing apoptosis and DNA damage for cancer therapy.
    Article Snippet: Bi-magnolignan (BM), a novel compound isolated from Magnolia Officinalis leaves, exhibits significant anti-tumor activity in vitro.. However, the underlying mechanism remains elusive.. This study examines the anti-tumor properties of BM and its mechanism of action, specifically through its interaction with BRD4, a key regulator in oncogene transcription and genome stability.

    Article Title: β-TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1.
    Article Snippet: Samples were analyzed by MS/MS using an LTQ mass spectrometer (Thermo Scientific, Waltham, MA, USA) as described previously [21].

    Single Cell Gel Electrophoresis:

    Article Title: Human RIF1-Protein Phosphatase 1 Prevents Degradation and Breakage of Nascent DNA on Replication Stalling
    Article Snippet: Mouse monoclonal anti-CtIP [D-4] , Santa Cruz , Cat# sc-271339; RRID: AB_10608728.

    Article Title: Discovery of Bi-magnolignan as a novel BRD4 inhibitor inducing apoptosis and DNA damage for cancer therapy.
    Article Snippet: Bi-magnolignan (BM), a novel compound isolated from Magnolia Officinalis leaves, exhibits significant anti-tumor activity in vitro.. However, the underlying mechanism remains elusive.. This study examines the anti-tumor properties of BM and its mechanism of action, specifically through its interaction with BRD4, a key regulator in oncogene transcription and genome stability.

    Article Title: β-TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1.
    Article Snippet: Samples were analyzed by MS/MS using an LTQ mass spectrometer (Thermo Scientific, Waltham, MA, USA) as described previously [21].

    Control:

    Article Title: Human RIF1-Protein Phosphatase 1 Prevents Degradation and Breakage of Nascent DNA on Replication Stalling
    Article Snippet: Mouse monoclonal anti-CtIP [D-4] , Santa Cruz , Cat# sc-271339; RRID: AB_10608728.

    Article Title: Discovery of Bi-magnolignan as a novel BRD4 inhibitor inducing apoptosis and DNA damage for cancer therapy.
    Article Snippet: Bi-magnolignan (BM), a novel compound isolated from Magnolia Officinalis leaves, exhibits significant anti-tumor activity in vitro.. However, the underlying mechanism remains elusive.. This study examines the anti-tumor properties of BM and its mechanism of action, specifically through its interaction with BRD4, a key regulator in oncogene transcription and genome stability.

    Article Title: β-TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1.
    Article Snippet: Samples were analyzed by MS/MS using an LTQ mass spectrometer (Thermo Scientific, Waltham, MA, USA) as described previously [21].

    Luciferase:

    Article Title: Human RIF1-Protein Phosphatase 1 Prevents Degradation and Breakage of Nascent DNA on Replication Stalling
    Article Snippet: Mouse monoclonal anti-CtIP [D-4] , Santa Cruz , Cat# sc-271339; RRID: AB_10608728.

    Article Title: Discovery of Bi-magnolignan as a novel BRD4 inhibitor inducing apoptosis and DNA damage for cancer therapy.
    Article Snippet: Bi-magnolignan (BM), a novel compound isolated from Magnolia Officinalis leaves, exhibits significant anti-tumor activity in vitro.. However, the underlying mechanism remains elusive.. This study examines the anti-tumor properties of BM and its mechanism of action, specifically through its interaction with BRD4, a key regulator in oncogene transcription and genome stability.

    Article Title: β-TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1.
    Article Snippet: Samples were analyzed by MS/MS using an LTQ mass spectrometer (Thermo Scientific, Waltham, MA, USA) as described previously [21].

    Sequencing:

    Article Title: Human RIF1-Protein Phosphatase 1 Prevents Degradation and Breakage of Nascent DNA on Replication Stalling
    Article Snippet: Mouse monoclonal anti-CtIP [D-4] , Santa Cruz , Cat# sc-271339; RRID: AB_10608728.

    Article Title: Discovery of Bi-magnolignan as a novel BRD4 inhibitor inducing apoptosis and DNA damage for cancer therapy.
    Article Snippet: Bi-magnolignan (BM), a novel compound isolated from Magnolia Officinalis leaves, exhibits significant anti-tumor activity in vitro.. However, the underlying mechanism remains elusive.. This study examines the anti-tumor properties of BM and its mechanism of action, specifically through its interaction with BRD4, a key regulator in oncogene transcription and genome stability.

    Article Title: β-TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1.
    Article Snippet: Samples were analyzed by MS/MS using an LTQ mass spectrometer (Thermo Scientific, Waltham, MA, USA) as described previously [21].

    Software:

    Article Title: Human RIF1-Protein Phosphatase 1 Prevents Degradation and Breakage of Nascent DNA on Replication Stalling
    Article Snippet: Mouse monoclonal anti-CtIP [D-4] , Santa Cruz , Cat# sc-271339; RRID: AB_10608728.

    Article Title: Discovery of Bi-magnolignan as a novel BRD4 inhibitor inducing apoptosis and DNA damage for cancer therapy.
    Article Snippet: Bi-magnolignan (BM), a novel compound isolated from Magnolia Officinalis leaves, exhibits significant anti-tumor activity in vitro.. However, the underlying mechanism remains elusive.. This study examines the anti-tumor properties of BM and its mechanism of action, specifically through its interaction with BRD4, a key regulator in oncogene transcription and genome stability.

    Article Title: β-TrCP overexpression enhances cisplatin sensitivity by depleting BRCA1.
    Article Snippet: Samples were analyzed by MS/MS using an LTQ mass spectrometer (Thermo Scientific, Waltham, MA, USA) as described previously [21].



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    Image Search Results


    Journal: Cell reports

    Article Title: BLM has Contrary Effects on Repeat-Mediated Deletions, based on the Distance of DNA DSBs to a Repeat and Repeat Divergence

    doi: 10.1016/j.celrep.2020.01.001

    Figure Lengend Snippet:

    Article Snippet: Mouse monoclonal anti-CtIP , Active Motif , Cat#61141, RRID:AB_2714164.

    Techniques: Recombinant, Reverse Transcription, Protease Inhibitor, Western Blot, Mutagenesis, Plasmid Preparation, Expressing, Software